Elongated fibres (fibrils) of the beta-amyloid protein form the typical senile plaque present in the brains of patients with Alzheimer’s disease. A European research team, working in conjunction with a team from the United States, has now succeeded in explaining the structure of the beta-amyloid peptide 1–42, which is the most important to the illness, at atomic resolution. This simplifies the targeted search for medicinal products to treat Alzheimer’s dementia.
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